Tuesday, March 31, 2015

Diabetes Management through Nutrition

 Diabetes Management through Nutrition


Prevalence

         Increases with age
         Gender Difference
         Racial, Ethnic – Disproportionate prevalence among African Americans, Hispanic Americans and American Indian



Increase in Overall Prevalence

         Increasing Age of US population
         Reduction in Mortality Rate
         Increase in Risk Factors such as
       Obesity
       Physical Inactivity



Mortality Risk
         Duration of Diabetes
         Lack of Blood Glucose Control
         Cardiovascular Risk Factors such as
       Smoking
       Hypertension
       Abnormal Lipid Levels
       Physical Inactivity
       Central Obesity



Causes of Death Among People With Diabetes












Geiss LS et al. In: Diabetes in America. 2nd ed. 1995:233-257.


Pathophysiology

         Genes
         Obesity
         Sedentary Lifestyle
         Aging


Classification
         Diabetes Mellitus and Other Categories of Glucose Intolerance
       DM (with four subclasses)
       Impaired Glucose Tolerance
       Gestational DM

Four Types of DM

         Type I DM (Insulin Dependent)
         Type II DM (Non-Insulin Dependent)
         Secondary/other types of diabetes associated with certain conditions
         Malnutrition related DM


Type I DM

         Presence of ketosis
         Almost complete lack of insulin or severe lack of
         Autoimmune Cause
         Patients commonly lean


Type II DM
         Most Common
         Strong Genetic Basis
         Absence of Ketosis
         Inadequate Insulin Secretion
         Obesity a strong factor


Secondary/Other Type

         Related to certain diseases, conditions or drugs
         Known or probable cause
         Treatment of underlying disorder may ameliorate the diabetes
         Hyperglycemia present at level diagnostic of diabetes


Malnutrition Related Diabetes Mellitus

         Mostly in developing countries
         Among 10 to 40 year olds
         Hyperglycemia present without ketoacidosis
         Role of malnutrition as a causal factor is unknown.


Impaired Glucose Tolerance
         Higher than normal plasma glucose but lower than the diagnostic values for DM
         Precursor for Type II
         Only about 25% develop into type II and rest go back to normal
         Patients are more susceptible to macrovascular diseases. 


Gestational DM
         2-4% during second or third trimester
         Onset of DM with pregnancy
         More common in older women with family history of dm
         Higher chance of developing NIDDM

  
Types of DM














* growing incidence in adolescents


 Diagnosis of Diabetes

         Polydipsia – Increased thirst
         Polyuria – Increased frequency of urination
         Fatigue
         Polyphagia – Increased Fatigue
         Weight Loss
         Abnormal Healing
         Blurred Vision
         Increased occurrence of infections



Diagnostic Criteria

FPG = Fasting Plasma Glucose                                                                 RPG = Random Plasma Glucose                                                              OGTT = Oral Glucose Tolerance Test













FPG = Fasting Plasma Glucose                                                                 RPG = Random Plasma Glucose                                                              OGTT = Oral Glucose Tolerance Test




Risk Factors for Asymptomatic Patients

         Strong Family History
         Obesity
         Certain Races
         Women with previous GDM
         Previous IGT
         Hypertension or hypertriglyceridemia
         40 years old with any of the above



Goals of Treatment

         Alleviate symptoms
         Prevent complications
         Prevent progression of current complications
         Improve quality of life


ADA Goals of Treatment (cont.)











ADA Goals of Treatment (cont.)
         Before Meals
       normal                                  < 110
       goal                            90 - 130
         Peak Post Prandial
       normal                                  < 140
       goal                            < 180
         Bedtime
       normal                                  < 120
       goal                            110 -150


Insulins

         Rapid acting: Lispro (Humalog) 
         Short acting: Regular
         Intermediate: NPH or Lente
         Long-acting: human Ultralente
         Basal insulin: Glargine

 Insulin action










Nutrition

         Nutrition Therapy – The Most Fundamental Component of the Diabetes Treatment Plan
         Goals:
       Near Normal Glucose Levels
       Normal Blood Pressure
       Normal Serum Lipid Levels
       Reasonable Body Weight
       Promotion of Overall Health



Nutrition Therapy Diet Teaching

         Goal - independence; effective self-management.
         Include Family.
         Follow prescribed plan; accurate portions
         Never skip meals
         Concern - Alcohol
         Concern - Dietetic Foods


Nutrition Consult
         Conduct Initial Assessment of Nutritional Status
         Diet History, Lifestyle, Eating Habit
         Provide Patient Education Regarding
       Basic principles of diet therapy
       Meal planning
       Problem solving
       Developing individualized meal plan
       Emphasize one or two priorities
       Minimize changes from the patient’s usual diet


Nutrition Therapy
         Provide Follow-up assessment of the meal plan to
       Determine effectiveness in terms of glucose and lipid control and weight loss
       Make necessary changes based on weight loss, activity level, or changes in medication
       Provide ongoing patient education and support


Nutrition Goals for
    Type 1 ***                                                                                     *** Type 2































Nutritional Goals (con’t)
      Type I **                                                                                               **Type 2


















Dietary Management of Diabetes

         Maintain as near-normal blood glucose levels as possible by balancing food, insulin and exercise
         Achieve recommended serum blood lipid levels
         Provide energy intake to maintain or attain healthy weight
         Prevent and treat acute and long-term diabetes-related complications
         Enhance over all health


Weight Loss

         Improves Glucose Control
         Increases Sensitivity to insulin
         Lower lipid levels and blood pressure
         Corresponding lowering of the dosage of pharmacologic agents

For a Successful Outcome

         Modest Caloric Restrictions
         Spreading caloric intake throughout the day
         Increased Physical Activity
         Behavior Modification
         Psychosocial Support

Nutrient Components

         Protein*
         Fat*
         CHO*
         Sucrose and Fructose
         Nutritive Sweeteners
         Fat Replacements*
         Vitamins and Minerals

Protein Intake

         Small to medium portion of protein once daily
         12-20% of daily calories
         From both animal and vegetable sources
         Vegetable source less nephrotoxic than animal protein
         3-5oz of meat, fish or poultry daily
         Patient with nephropathy should limit to less than 12% daily

Fat Intake

         <35% of total calories
         Saturated fat <10% of total calories
         Polyunsaturated fats 10% of total calories
         Cholesterol consumption < 300 mg
         Moderate increase in monounsaturated fats such as canola oil and olive oil (up to 20% of total calories)

CHO Intake

         CHO intake determined after protein and fat intake have been calculated.
         Emphasize on whole grains, starches, fruits, and vegetables
         Fiber same as for nondiabetics (20g to 35g)
         Rate of digestion related to the presence of fat, degree of ripeness, cooking method, and preparation

Nutritive Sweeteners and Fat Replacements

         Nutritive Sweeteners: corn syrup, fruit juice concentrate, honey, molasses, dextrose, and maltose have same impact on calorie and glycemic response
         Fat substitutes are derived from CHO or protein sources.  So, CHO and Protein content should be reviewed before using
Nutrition
         Individualized Diet Treatment Plan
         Diet changes do not have to be dramatic
         Regular monitoring of blood glucose, glycated hemoglobin, lipid levels, blood pressure, and body weight

Exercise

         Potential Benefits
       Improved Glucose tolerance
       Weight loss or maintenance or desirable weight
       Improved cardiovascular risk factors
       Improved response to pharmacologic therapy
       Improved energy level, muscular strength, flexibility, quality of life, and sense of well being

Precautions and Considerations

         Consult a physician
         Rule out significant cardiovascular diseases or silent ischemia
         Prevent hypoglycemia with self-monitoring of capillary blood glucose (SMCBG) both before and after exercising
         Strenuous exercise not recommended for people with poor metabolic control and significant complications

Exercise Prescription

         Interest
         Capacity
         Motivation
         Physical status
         Individualized approach











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